Does Meat or Sugar Cause Gout? The Truth May Surprise You.

Does Meat or Sugar Cause Gout? The Truth May Surprise You.

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Doctors Can't Tell You What They Don't Know

Most Doctors don’t know anything of the information I am about to share with you and the science behind it is as completely contrary to what  doctors are taught.

In fact, after formal schooling, pharmaceutical sales reps are the ones who educate doctors on new drugs.

Maybe it’s time we take a fresh look at some old and new science that suggest that meats have little to anything to do with causing gout attacks. You have questions, and we have answers.

How does a disease manage to be misunderstood and nearly forgotten while still affecting millions of Americans each year? Gout does just that, right under our noses. For those affected by gout and the excruciating pain that comes with it, there is no way to forget it.

Gout has been around for centuries. In the time of castles and knights, it was called the “ailment of kings”. Then it only had an effect on the rich. The rest of the population could barely afford to eat, let alone eat to excess.

Now things are different. America is one of the heaviest countries in the world with Mexico moving just ahead of us. The rise in obesity in countries like Mexico is attributed to the increasing availability of sugary convenience foods. This may result in a rise in gout patients in the coming years.

Wait?! I thought gout was caused by eating too much fatty meat? Isn’t that what we’ve always known? As with other medical knowledge, our understanding is moving forward. We know more about what causes gout and how to treat it than we ever have. The hard truth is that over consumption of sugars is a major contributing factor in gout patients.

What Is Gout?

Let’s back it up a little and examine what’s going on here. First, what is gout?

Gout is a very specific kind of arthritis. In normal arthritis, joints swell up and cause discomfort. Gout is usually focused on a single joint, often the big toe. That joint fills up with sharp uric acid crystals. The joint responds by swelling up. The swollen joint is very tender to the touch, the pain often being described as a fire. Patients sometimes wake from their sleep, unable to stand even the gentle touch of their sheets.

Those with gout suffer because of too much uric acid in their blood stream. We all have some uric acid, and it doesn’t cause a problem for us. Gout patients have more than 6 or 7 mg/dl of uric acid in their blood. Uric acid levels have been recorded at and over 12 mg/dl.

The uric acid crystals that build up in a joint and cause gout pain can have other effects as well, including kidney stones and kidney damage. These high levels are found in over 20% of people in the US.

Uric acid occurs in your blood stream as a byproduct of the breakdown of purines. Purines are necessary and a vital building block for your cells. As with everything, there can be too much of a good thing.

For years, a low purine diet and medication has been the solution to gout. This is generally described as a diet with less meat and fewer high purine foods. High purine foods include more than just meat. They include meat products, yeast, beer and booze, beans, peas, oats, spinach, and mushrooms.

Low purine foods are said to include refined bread products like white bread, processed cereals, and pasta. Also included are sugary sweets, milk, eggs, butter, fruit, nuts, and carbonated drinks.

Eating Junk Food Doesn't Help

Wow, sounds like you can eat a bunch of junk food and get rid of your gout right? This is not necessarily the case. Keep reading.

It’s also pretty old news that drinking a bunch of alcohol can bring on gout symptoms. Partying too hard is definitely off your schedule when you have gout. This isn’t your doctor being a spoil sport, and the science backs this up. Just a few drinks can push your uric acid levels up significantly.

Could It Be... Sugar?

Sometime in the late 1960’s, doctors figured out that fructose (sugar found in fruits) can raise your uric acid levels as well. Fruit, sugar, and corn syrup all have fructose in them. Just like beer, consuming these things can push your uric acid levels up in a real way. If you already have gout, your response to fructose will be more severe.

This is when doctors also noticed that poor people were getting gout more and more often. We thought this was a disease for the rich right? Sugar and sugary foods became cheap and readily available to the poor, changing the demographic that gout targeted.

Fructose Turns To Fat In The Body

This all sounds like a lot of conflicting information, but it’s really a simple shift in what we knew. We can now better explain things scientifically than just with anecdotal data. The simple explanation, scientifically, is that fructose is a sugar that turns to fat easily.

When your liver is busy doing that, it needs a lot of a compound called ATP. ATP has purines, lots of them. They get turned into uric acid. Alcohol breaks down just the same way!

This gets worse. While the sugar is busy making sure your blood has uric acid, your body is releasing insulin. Insulin sends a message to the kidneys and get it to put uric acid right back into your blood. Insulin wants you to grow. To grow you need to make a bunch of cells. To make a bunch of cells, you need purines. It’s a simple chain reaction.

You Can't Cut Both Carbs AND Meat

So you have gout, and you’ve started that low purine diet, cutting out meat and booze. Now you’re eating a bunch of easily affordable cheap carbohydrates in place of all that meat you were eating. Then you find out the sugar contributes to your gout even more! Do you cut out carbohydrates and meats? How does that work nutritionally? Very simply, it doesn’t!

There are plenty of cultures that consume a ton of meat, but have no problems with gout. If you look historically, before crops were as reliable as they are now, vegetables were not guaranteed. This meant that meat was necessary in every diet.

While meat is high in purines, protein is the thing that helps lower purines. To an effect, meat can clean up after itself in your bloodstream. Meat cannot clean up after itself AND a bunch of sugar though.

Studies that were just paying attention to meat consumption did not take into account that people eating more meat were also eating more of everything else too. They did not measure carbohydrate intake at all. This is how science sometimes misses something.

But, Where's The Research?

Real, clinical studies for gout are almost non-existent, and there are none that deal with what we’re talking about. Studies we have began to focus on medication, and the best study that dealt with diet (in Brazil) did not use actual gout patients.

A teeny tiny study was done exploring gout and sugar. In 2009, a few men with gout were chosen in South Africa. These men were put on a 1600 calorie diet. Purines were not measured, and alcohol was allowed. The diet was 40% (not refined) carbohydrates, 30% protein, and 30% fat. Four servings of fish a week kept their protein a little lean. The study lasted 16 weeks.

Uric acid levels went down, on average, 18%. Seven men left the study with a normal uric acid level. Gout attacks were reduced by 72% and 17lbs were lost.

Because dietary changes were sweeping, it’s hard to know which changes affected the patients the most, which is why more widespread studies are needed.

What kind of meat did they eat while trying to stay away from unsaturated fats? What were their diets like before the study began? These are details that could help us.

Fructose Contributes To Gout More Than Alcohol

Another study done on gout confirms that sugar intake, especially fructose, can be a major contributing factor in gout symptoms.

In a study of 51,529 professional males, a study of dietary habits monitored the possibilities of what could cause gout in men from ages of 40 to 75 years old during the years of 1986 to 1998. Only 49,166 participants mailed in their evaluations the first year of the study.

These men were given evaluation sheets to turn in of what they ate and didn’t eat by marking pre-assigned amounts on the sheets. They were dentists, optometrists, veterinarians, osteopaths, and pharmacists.

The study also accounted for the effects on any exercise, medication, or medical conditions that the participants might have had prior to starting the study. This also studied any changes in the participants exercise, medication, or developments of medical conditions during the study.

Along with keeping up with the BMI in the participants over the years. During this study, there were 755 newly diagnosed cases of gout in the participants. Participants that reported having a diagnosis of gout on a previous evaluation forms were dropped from the study.

Only 2,773 participants reported having gout at the beginning of the evaluations. Out of these participants, 50 medical records were evaluated by medical professionals to see if they had 6 out of 11 indicators for gout. Then they were checked to see if the diagnosis was self diagnosed, diagnosed with a doctor, or reported with a tophus or crystal proved gout.

The evaluation sheets were tabulated every 4 years, to see the progress of the study’s participants. The participants in the study could miss one report or check in for the study one year for a follow up evaluation, and turn in the sheets during the next evaluation. There was a follow up rating of over 90% for the two year period.

The study came out that the urate, found in gout patients tended to raise in participants that had instances of high fructose consumption. Such as soft drinks and fruits with high sugar levels. Diet soft drinks did not have the same rates as soft drinks had evaluated with.

Surprisingly, alcohol or spirits weren’t as high in causing gout as fructose, with results being 35% and 49% for soft drinks with sugar and 15% for beer and spirits.

The study also evaluated the effects of dairy, alcohol, and BMI as risk factors for developing gout. These proved to have no significant impact on the study findings. Also measuring the effects of carbohydrates (non-Fructose) and protein in regards to the energy produced in junction with Vitamin C. Even with all these variables, the study proved that high intakes of fructose and soft drinks caused higher levels of urate crystals in the joints.

From the data obtained during this study, it gives credibility to Dr. Osler’s prescription of low fructose diets for gout patients over 100 years ago. In which he wrote in 1893 that high sugar diets should be reduced to a minimum, and sweeter fruits should not be eaten.

There are limitations to this study, even though it was the highest participant based study to this date, due to the fact that dietary intakes were reported by the participants data, though a sampling of the evaluations further confirmed the results.

Gout Is Associated With Metabolic Syndrome

Another study was conducted that confirmed Dr. Osler’s ideas. This study was done over a slightly longer period of time, but it echos some similar thoughts. It also was opened up to include women, something not done in previous studies.

The nurses at American College of Rheumatology did this study to examine the relationship between high intake of fructose rich beverages and risk of gout. The study was 22 years long (1984 to 2006) and had 78,906 participants, who previous to the study had no history of gout.

The all had to take a food frequency questionnaire to validate their intake of beverages and fructose. During the 22 years, the follow up the nurses documented 778 confirmed cases of gout. The test results showed that, in women, the more sugary soda consumed, the higher the risk of gout is. Diet soda drinks were not corresponding to the study.

New evidence shows that gout is associated with the metabolic syndrome and may lead to a higher risk of heart attack), type 1 and 2 diabetes, and premature death. Metabolic syndrome is a cluster of conditions — increased blood pressure, a high blood sugar level, excess body fat around the waist and abnormal cholesterol levels — that occur together, increasing your risk of heart disease, stroke and diabetes.

Gout studies and treatments have been long focused on men, but there is new evidence that suggest that it is more commonly found in elderly women over 70 years of age. The confirmed gout cases are beginning to become more frequent in women.

Sugar sweetened beverages contain low levels of purine, the precursor of uric acid, however they contain large amounts of fructose, a carbohydrate known to increase uric acid levels. When the uric acid levels rise it causes frequent attacks of gout, or it may never cause problems. A high uric acid level may also cause some people to develop kidney stones or kidney failure.

In this study, ingesting fructose was found to cause a sharp increase in serum uric acid. The rapid increase was exaggerated in individuals with a history of gout. Also, in a different study it was found that animal experiments and two National Health and Nutrition Examination Survey (NHANES) studies have suggested that the enormity of urate raising effect of sugar sweetened sodas may be weaker among women than among men, extrapolation of data on this important risk factor for gout from men to women should be done with caution.

More Validation of the Sugar Connection

The Nurses’ Health Study (NHS) began in 1976 when 21,700 female RNs who were predominantly white and were 30 and 55 years of age living in 11 different states completed a mailed questionnaire in which they provided detailed information about their medical history, lifestyles, and other risk factors. Their information was updated every two years to try to help identify any other risks, and newly diagnosed diseases.

After the women completed the questionnaire, they excluded women with a previous diagnosis of gout before 1984 or the participants who did not complete more than 10 items on the 1984 dietary questionnaire, leaving 78,906 women who were followed from 1984 to 2006.

The Partners Healthcare System institutional review board approved the study. The review board accepted a return of the completed questionnaire as implied informed consent.

The questionnaire consisted of average use of foods and beverages during the previous year, including sodas, juice, and the food that the participants ate. The NHS based their study on the average of products consumed from the questionnaire.

The Connection With Soda & Juice

The nutrient intakes were computed by multiplying the responses by the nutrient content of the specified portion sizes. The values for nutrients were derived from the US Department of Agriculture, and supplemented with information from manufacturers of the products consumed.

Half of the disaccharide sucrose is fructose, which is split from sucrose in the small intestine. So, total fructose intake is equal to the intake of free fructose plus half the intake of sucrose. Knowing this, the relationship between the questionnaire and multiple dietary records showed that the most consumed drinks were soft drinks and fruit juices.

After every two years, the participants provided information on weight, regular use of medication, and medical conditions. This data has proven to be very helpful in the study, validating the study, and have the ability to predict risk of relevant future diseases.

The questionnaires asked the participants had received a physician diagnosis of gout and, if so, the date of first occurrence. In 2001, NHS sent out a questionnaire to the participants with self diagnosed gout, and asked them to go to their physician and get a confirmed diagnosis.

The end point of the study was an occurrence case of gout in which 6 or more of 11 gout criteria were met. The gout criteria are as follows: more than one attack of acute arthritis; maximum inflammation developed within one day; oligoarthritis attack (arthritis affecting one to four joints during the first six months of disease) redness observed over joints; painful or swollen fingers, toes, or joints; tophus (a deposit of crystalline uric acid and other substances at the surface of joints or in skin or cartilage); hyperuricemia (an abnormally high level of uric acid in the blood); asymmetric swelling within a joint; complete termination of an attack.

The overall rate for gout questionnaire was 81%, which was a similar result to the Health Professionals Follow up Study. In this study two Board certified rheumatologists reviewed the medical records from 56 women in 2001. The similarities between the diagnosis of gout and the review of the relevant medical records was 91% (51/56).

Diet Sodas Don't Seem To Affect Gout Risk

In the study they used Cox proportional hazards modeling (PROC PHREG) to estimate the relative risk (RR) for occurrences in gout. They put consumption of soda and juices into six categories: <1 per month, 1 per month to 1 per week, 2 to 4 per week, 5 to 6 per week, 1 per day and 2 or more per day. Fructose intake were categorized into quintiles for percentage of energy.

Soda and juice consumption were adjusted for the following variables: age (continuous), total energy intake (continuous), alcohol consumption, body mass index (kg), menopause status, use of hormonal replacement, use of diuretics (thiazide or furosemide), history of hypertension, coffee intake, and daily mean intake of meats, seafood, dairy foods, and total vitamin C (quintiles).

They did the same for fruit and non dietary products. (alcohol, etc.). During the 22 years of follow ups the found 778 new diagnosed cases of gout, 638 with podagra (gout of the foot, especially the big toe),576 with hyperuricemia (abnormally high level of uric acid in the blood), 342 with tarsal joint involvement, and 109 with tophus (deposit of crystalline uric acid and other substances at the surface of joints or in skin or cartilage).

In this large study of women, it was found that the risk of gout increased with increasing intake of sugar sweetened soda. However, diet soda intake does not have any risk of gout. The study shows that women who consume 1 serving of sugar sweetened soda have a 74% higher risk of getting gout than women who consume two servings of orange juice.

The study shows the risk of gout was significantly increased with increasing intake of fructose, the main suspected ingredient behind the increased risk. These findings were separate from risk factors for gout such as body mass index, age, hypertension, menopause, diuretic use, alcohol, and intake of dairy, meat, seafood, coffee, and vitamin C.

These findings were confirmed by the recent prospective study of men and provide the first evidence among women that fructose and fructose rich beverages are important risk factors to be considered in the prevention of gout.

Gout Among Women Occurs Largely After Menopause

While the risk for gout associated with the fructose rich beverages, absolute risk differences were modest given the low occurrence rate of gout among women. The risk differences were less than one case per 1,000 person years.

Previous animal experiments and NHANES studies suggest that the high quantity of urate raising effect of fructose or sugar sweetened sodas may be weaker among females than among males. An analysis based on NHANES III found that the increase in serum uric acid level associated with sugar sweetened soda intake was larger among men than women, although the association among women was still significant.

This was thought to be because of sex hormones due to study in rats that have shown that female sex hormones protect against the development of hyperinsulinemia (a condition in which there are excess levels of insulin circulating in the blood than expected relative to the level of glucose) associated with high fructose intake.

Nevertheless, as gout among women occurs mainly after menopause, when the female hormonal influence greatly declines, the gender difference of the fructose effect on the risk of gout may be less apparent than that on serum uric acid levels observed in the general population that included premenopausal women.

Fructose Causes Uric Acid Production

Fructose induces uric acid production by expanding ATP (a nucleotide that is composed of adenosine and three phosphate groups and releases energy when hydrolyzed to ADP) and deteriorating it into AMP. Fructose in the liver uses ATP, and the accompanying phosphate deterioration limits the regeneration of ATP from ADP (an ester of adenosine that is converted to ATP for the storage of energy) which serves as surface for the catabolic pathway to uric acid formation.

Therefore, within minutes after fructose infusion, plasma uric acid concentrations are increased. In opposition, glucose and other simple sugars do not have the same effect. Fructose could indirectly increase serum uric acid level and the risk of gout by increasing insulin resistance and circulating insulin levels.

Due to animal experimentation, from short term feeding studies suggest that higher fructose intake provides to insulin resistance, impaired glucose tolerance, and hyperinsulinemia. The NHS studies have practical instructions for the prevention of gout in women.

They have dietary recommendations and restrictions on purine intake. Low purine diets are often high in carbohydrates including fructose rich foods. Their data stresses the importance of restricting fructose intake. Osler’s diets prescription as a means to prevent gout over 100 years ago, as reflected in his 1893 book supports this study.

Also, fructose intake is related to increased serum insulin levels, insulin resistance, and increased adiposity. The general negative health impact from fructose is larger in women with a history of gout, 70% of those women suffer from the metabolic syndrome.

There were a relatively large number of cases in the high fructose groups. The numbers in the top consumption categories of fructose rich beverage items were small. It was comforting that the next top categories also showed large positive associations with a dose response relationship. Biased recall of diet was avoided in this study because the intake data was collected before the diagnosis of gout.

Gout Risk Is Higher For African Americans

The use of repeated dietary assessments in the study not only accounts for changes in dietary consumption over time but also lowers measurement error.

The restriction of RNs in the study is both a limitation and a strength. The study of well educated women reduces the potential for surprises associated with socioeconomic status, and they were able to obtain high quality data with minimal loss to the follow ups.

Their study was generalized towards middle aged white women with no history of gout. Prevalence of risk factors for gout occurrences are higher in African Americans, the enormity of the risks increase with the sugar sweetened beverages might be bigger than the increase that they observed.

In conclusion, their findings provided the prospective verification that intake of sugar sweetened sodas, orange juice, fruit juice, and other fructose is associated with an expanded risk of gout among the participated women.

Their contribution to the study and risk of gout in the population is likely humble giving the low occurrence rate among these women. However, diet soda consumption was not associated with this study or the risk of gout.

Physicians should acknowledge of the significant impact of these sugar sweetened sodas and fruit juices on the risk of gout which is a common and excruciatingly painful arthritis.

We Were Never Meant to Eat This Much Sugar

So how does all of this affect you, a member of normal society living long past the age of kings? Well, lay off the booze. It is consistently a problem for gout across the board, so it’s an easy change to make. Even a few beers can throw you way off track, so exercise some self discipline.

Research doesn’t know it all yet. We look very hard to doctors and science for our cures, but when their information is incomplete, we can take some steps on our own and see what works for us as individuals.

First, this means knowing your diet. Record what you eat, and record when you get your gout attacks. You might find a pattern there. Self awareness is the first barrier of defense we have against something wrong medically.

We don’t know why some things happen, even when we know that they do happen. We don’t know why only some people with high uric acid levels end up with gout. Saying that we don’t know might seem scary. It’s hard to not know something in an age where there’s an answer for everything, but science continues to push forward. This means that we have to get up and help ourselves where we can.

Take the science that you know and add in some of your basic common sense. If you have a bad diet, you will suffer the consequences of a bad diet. Purines come in our natural food and are produced in our bodies.

Cut The Carbs & Check Your Blood Sugar

No, you don't have to go THIS far!

While you’re making a record of your diet and your gout attacks, record other medical data too. Knowing your blood sugar is a good thing, and meters are readily available. Sometimes meters are even free, and your doctor is unlikely to object to this. Doctors want you to be as aware medically as you can be so that you can take responsibility for your health.

Cut out refined sugars like white bread, sugar cereals, table sugar, sodas, candy, and junk food. Then wait and see what happens, recording the results. Do your gout attacks decrease? How do your blood sugar levels fare? Remember to follow the rules you set for yourself. You are acting as your own scientist, and deviating from your plan will alter your results. Consistency is the path to change.

If nothing else, your doctor will be thrilled as your health gets better overall. There are other small things you can do too. Taking Vitamin C has been shown to reduce uric acid just a little, so pop 500mg a day of it.

Make sure you replace those refined carbs with some good stuff. Whole grains and yummy vegetables will fill your meals back out. Cauliflower is amazing, and people are learning to use it in place of potatoes in all kinds of dishes. It can be steamed and turned into a substitute for rice too! In a food processor with a bit of butter and some plain yogurt, it turns into a substitute for mashed potatoes that’s as good as the real thing.

Don’t take your produce section for granted, and don’t just use your computer for games. Recipes low in refined carbohydrates are easily found online, leaving you with very few excuses not to change your health for the better, staving off a painful disease in the process.

Gout was once an ailment only felt by royalty. It now effects millions of people worldwide, and our knowledge is expanding on the subject.

Diet Is The Ultimate Solution

It’s becoming clear that the solution for many of our illnesses begins with our diet. Don’t eat blindly. Instead, take into account what you are eating and what went into making your meal.

To be effective you need to understand more about diet and have some good recipes or you will fail.

The good news? I already failed enough for all of us and have been gout attack free for over 5 years.

I figured you may want to know how I stopped my extremely vicious battle with gout.

Here are my guides (cookbooks) below that help me eat right for someone who suffers from gout. It cuts carbs (sugars) and my gout just magically disappeared.

You only need to pick one to get started, but having more gives you more variety.

Cherry Juice For Gout: The Complete Story

Cherry Juice For Gout: The Complete Story

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Do a little word association. If I say the word “gout”, what do you think of? Do you think of old men or pork? You probably don’t hear much about gout, though it effects millions of people each year.

Specifically, over 8 million people have gout. In 2008 alone, 8.3 million people reported that they had been diagnosed by their doctors with gout! While gout is considered to predominately effect men, the numbers become much more even after women reach menopause. After that time, the rate of gout in men vs. women is almost even.

So what exactly is gout? Gout is an incredibly painful form of arthritis caused by a build up of uric acid crystals in the bloodstream. It usually effects the big toe, but can cause pain in other joints as well.

Uric acid is naturally found in our bloodstreams, but when too much of it builds up, it forms into crystals. Those crystals build up in the big toe, causing debilitating pain.

The pain is often described as a fire, and sufferers report not even being able to have a sheet over their toes when a gout attack hits. To make matters worse, flare ups traditionally wake patients from their sleep because they occur most often in the middle of the night.

To understand how to treat gout, we have to understand how it got started in the first place. While genetics play a part, lifestyle factors are largely to blame for gout symptoms. Meat is traditionally blamed for gout, because it effected mostly the ruling classes in earlier times.

Now we know that sugar and alcohol are more damaging and more likely to cause gout. The best treatments, in addition to lifestyle changes, come in a combination of homeopathy and scientific medicine (I have made it over 3 years without pharmaceuticals, so stick with me if that is your goal).

One of your secret weapon at home: cherries.

Don’t use just any cherries though. The type of cherry can make all the difference. So which cherries do you eat? Is it better to have the whole fruit or the juice? Are supplements as effective? Let’s talk about those things.

What Causes Gout?

So you know the basic causes of gout, but how does your body go from taking in too much sugar to having your big toe feel like it’s on fire? We’ll break that down now in plain language.

The foods you eat are broken down into enzymes and proteins. Your body funnels these where they need to go, and then your body uses them as fuel to run or building materials. When you eat more than you need of something, your body has to do something with it.

With water soluble things, you excrete them through your urine. The process from food to gout is begun with purines. The purines are filtered through your kidneys. When your kidneys get too much to filter properly, they end up in your bloodstream as uric acid. Different foods contain different amounts of purines.

While the science is not exact, sugar jacks these levels way up. This includes sugar from fruit, soda, and alcohol. All that uric acid can’t just hang out in your bloodstream, so the bits of it bond together into crystalline structures that have sharp edges. The sharp edges in these crystals cause them to stick together. They get all clumped up in your big toe, and your body responds as it would to any foreign object. The big toe swells. The more swelling that happens, the more those crystals stab.

The resulting redness and burning is incredible. A gout attack can last a few days up to over a week. After it goes away, it might be a few weeks, a few months, or even a few years before you experience another one. The goal, obviously is to lessen the time an attack lasts and to extend the time between attacks.

How Cherries Help With Gout

So now you know how your food can turn into gout. The next question is: how do we stop it? Let’s talk about how that secret weapon can reduce your gout symptoms and lengthen the time between attacks.

Scientists have been astounded by what they’ve learned about a simple cherry’s ability to help gout patients. Recent studies involving hundreds of patients concluded that gout sufferers who at one cup of cherries every day reduced their likelihood of another attack by 35%. More cherries equaled a lower risk of attack.

How do cherries help? I told you earlier that the foods you eat are broken down into parts. When cherries are broken down, your body puts parts like bioflavonoids and anthocyanins to work. Simply, these things fight inflammation and lessen uric acid levels in the blood.

Less uric acid in the blood means less work your kidneys have to do, keeping the buildup of uric acid down and eliminating those harsh, sharp crystals.

It takes as few as two servings of cherries every day to get your uric acid down a significant amount. Doing this over a long period of time can help you keep steady control over your uric acid levels, all but eliminating symptoms when combined with other pro-gout foods and foodstuffs like apple cider vinegar.

What makes cherries that one magic bullet though? Don’t other fruits have the compounds that can help your gout? Yes and no. First, let’s get something clear. There are no magic bullets for gout. A change in lifestyle is the only sure method to winning this fight, but back to the cherries because they do help from my personal experience.

Anthocyanins 1 and 2 are found in the best concentration in cherries. Other fruits usually only contain one of the two types. Cherries also pack bioflavonoids and antioxidants too, making them basically a gout super fruit.

Bioflavonoids act as both an antioxidant and as an anti inflammatory agent. Antioxidants in cherries help prevent uric acid from building up.

All these things together make cherries perfect, but what kind of cherries are the best for gout?

Sweet Cherries vs Tart Cherries For Gout

When you talk about cherries and gout, you hear two types of cherries first: tart and sweet: Comparing the two can help you decide what’s best for your body.

Tart cherries have much high concentrations of anthocyanins, three times as much in fact. The anthocyanins are what give tart cherries their deep, rich color and their healing abilities.

While sweet cherries are much tastier than their tart friends, you have to eat more of them to help with your gout. More sweet cherries means eating more sugar, and we discussed that sugar is one of the things that is causing your gout in the first place.

Making tart cherry juice drinkable is the biggest hurdle. In juice form, you can mix it with other things to enhance the flavor, which tart cherries on their own are harder to work with.

Try mixing your tart cherry juice in with some green tea or sparkling water. Much like cranberries, the flavor is strong, so it can be used as a flavoring for beverages.

The Black Cherry Extract

Another things you might consider is black cherries. While they are not a replacement for tart cherries, they can provide protection against gout symptoms as well. Black cherry extract is a great source of antioxidants and is well known to help joint pain.

Black cherry extract also helps process the uric acid in your system, and you want to do anything you can to keep that uric acid from building up and crystallizing.

It’s common to take one or two doses of black cherry extract three times per day, but talk to your doctor first. Consulting your physician can head off any problems you might have. It also allows you to scientifically track your progress so you know what is helping and how much.

Cherry Extract Pills & Supplements

If drinking cherry juice is just too much for you, pills and supplements are available. While they are helpful, following instructions and measuring doses is incredibly important here!

The first thing to consider is the brand you choose. Many brands of supplements have recently been accused of making fraudulent claims as to what is in their pills. Independent testing revealed the brands using filler products instead of the listed ingredients. When in doubt, do your research and go with a brand that you trust.

The second thing to consider is an appointment with your doctor. Bring your research and a list of questions with you to the appointment. Let your doctor know that you’d like to start taking the supplements, and get an opinion.

This will do several things for you. First, if you have any allergy or negative reaction, your doctor will know where it’s coming from. Secondly, you can track your progress medically through tests, which lets your assess your progress scientifically to tailor the best program for you.

Homeopathy should always be used alongside the medical advice and treatment of your doctor. You should also use it with the same care you use when your doctor gives you instructions. If you have a prescription that tells you how much and when to take a medicine, you follow those instructions. Homeopathic supplements should be taken with the same care and discipline to be effective.

With supplements, consider dosage and efficacy when choosing a product. Also remember that dried cherry in a supplement will take longer to digest than cherry juice, and you should allow for that time.

Combining Homeopathic Medicine And Traditional Medicine For Gout

There is not one simple magic pill to cure your gout. Different things help in different amounts. It varies person to person. You want to attack the cause of the problem, the symptoms, and the disease itself. Cherries help a lot with these things, but they may not be enough for you.

I want to warn against adding things to your routine without thought, research, or discipline. Just pouring some cherry juice now and again will not help you, and it will likely put you off of finding help for your gout. Anything you take, use a measured dose at regular intervals. Use the same discipline you’d apply to traditional medication.

Your doctor can test the uric acid buildup in your blood. If your doctor knows that you are going to start a line of treatment like cherries, they can test your baseline uric acid levels.

They can also provide regular testing to see how much the cherries are helping. This allows you and your doctor to work as a team, adjust your dosages, and find exactly what works for you.

Your doctor can also provide Cox-2 inhibitors via certain NSAIDS. Cox 1 and 2 are enzymes that promote inflammation. An inhibitor, found in some NSAIDS, can reduce the inflammation and keep it down.

There are many things you can use to help manage pain and inflammation. Use cherries, apple cider vinegar, eliminate alcohol and as much sugar as you can from your diet and you will see marked improvements.

 

Your Diet Is The Number 1 Way To Stop Gout Attacks Before They Start

It’s really strange that most do not take diet seriously, but given the (excuse the language) crap that is out there, even main stream science based medicine is full of hogwash.

What you need is a cookbook that gives you what you need to eat and how to prepare it. Let’s be frank, most will not change their diet if it taste like 20 year old cracker that was lost in the pantry. But the book I am suggesting is not like that. It made it easy for me to not only change my diet, it made me change my mind about food in general. You can find this book here. I promise, you’ll thank me later.